Philosophically, I am not satisfied by the doctor
saying, no Parkinson yet, pointless to worry about a disease with no medicine,
only symptomatic medicine! I am not like 99.9% who take it lying down! At the
same time, I chase only philosophically valid (have untested but possible
rationale) wind mills and never mills that defy current medicine. In other
words, attribute innovations filling completeness only. Parkinson is better than
similar Alzheimer’s being more visible, even then an elderly disease.
Why believe
US MRI and MRA?
Taking sections of my brain images, specially the
substia-nigra part, normal people have dopamine cells appearing black. These
are progressively reduced. That is what has happened to me, even though current
symptoms are not debilitating. Why? Don’t care yet!
How do
doctors handle it?
Symptomatic. There are 3 steps on any disease.
1.
Stop the
damage.
2.
Restore the
chemicals not being produced.
3.
Restore the
damaged part.
The doctors are helpless in 1 and 3. L-dopa is given
for 2. It works for 5 years. By then the body gets used and low effective even
as disease progresses. Switch to others for another 5 years. By then the
patient has progressed to dementia!
So how do
you expect to be different?
Parkinson’s disease involves gradually worsening tremors and movement problems. It is thought to be caused by a protein called synuclein found in nerve cells folding into the wrong shape, which triggers a chain reaction of misfolding in nearby synuclein molecules. This leads to a build-up of long strands or “fibrils” of the protein, killing neurons.
By nature I consider homeopathy criminal, Ayurveda rarely applicable, allopathy dumb half the time. The applicable part is modern medicine, mm. Most doctors don’t educate themselves in modern practices and are routinely dumb, especially against diseases on elderly and aging. Not surprisingly, Parkinson and Alzheimers resemble Aging, may have common solutions outside the comprehension of mm doctors. I believe we have discovered anti-aging in nmn, anti-debilitation in Mitoq and metformin for pseudo-diabetes from aging. Time will tell. I do believe that my anti-aging program will make me first human to attack Parkinson and Alzheimer’s and make survival to age 150 worthwhile, awaiting transfaction based extension to tghousands of years!
By nature I consider homeopathy criminal, Ayurveda rarely applicable, allopathy dumb half the time. The applicable part is modern medicine, mm. Most doctors don’t educate themselves in modern practices and are routinely dumb, especially against diseases on elderly and aging. Not surprisingly, Parkinson and Alzheimers resemble Aging, may have common solutions outside the comprehension of mm doctors. I believe we have discovered anti-aging in nmn, anti-debilitation in Mitoq and metformin for pseudo-diabetes from aging. Time will tell. I do believe that my anti-aging program will make me first human to attack Parkinson and Alzheimer’s and make survival to age 150 worthwhile, awaiting transfaction based extension to tghousands of years!
What differences
in your routine?
Parkinson is basically mitochondria based disease. For
it to be helped, you must essentially do to brain-cells what is done for anti-aging
outside, namely nmn. That crosses the blood-brain barrier. Neither ECP or HOT
will help. How can one do ECP on brain fluids?One great opportunity is in quantum dots in brain, possible in 5 years! Note that elderly diseases of Alzheimer's (amyloids)and Parkinson's (synclein) are both caused by protein aggegates, attacked by the quantum dots.
Nature Nanotechnology (2018) |
Abstract
Though emerging evidence
indicates that the pathogenesis of Parkinson’s disease is strongly correlated
to the accumulation1,2 and transmission3,4 of α-synuclein
(α-syn) aggregates in the midbrain, no anti-aggregation agents have been
successful at treating the disease in the clinic. Here, we show that graphene
quantum dots (GQDs) inhibit fibrillization of α-syn and interact directly with
mature fibrils, triggering their disaggregation. Moreover, GQDs can rescue
neuronal death and synaptic loss, reduce Lewy body and Lewy neurite formation,
ameliorate mitochondrial dysfunctions, and prevent neuron-to-neuron
transmission of α-syn pathology provoked by α-syn preformed fibrils5,6. We observe, in vivo,
that GQDs penetrate the blood–brain barrier and protect against dopamine neuron
loss induced by α-syn preformed fibrils, Lewy body/Lewy neurite pathology and
behavioural deficits.
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